By Gregory R. Bock, Julie Whelan (editors)
Records the newest effects and evaluations at the explanations and attainable therapies for this illness. assurance comprises retroviral involvement, immunity, pathophysiology and pharmacological remedy of continual fatigue syndrome.
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Extra resources for Chronic fatigue syndrome
Differences in the prevalence of somatization, for instance, may be entirely explicable, and expected, on the basis of this difference in the sample. Secondly, a crucial issue in your work, and for us all, is how you decide to ‘explain’ a fatigue case on the basis of major depression or of CFS, when the diagnostic criteria overlap so greatly. How do you make that attribution? Manu: I don’t make the attribution. These patients fulfil criteria for the CFS; at the same time, 75% of them fulfil criteria for major depression.
If you are going to make the point that physicians in general under-diagnose somatization disorder, that’s a valid point. It’s a bit more dangerous to say that people who study chronic fatigue are exposed to more somatization disorder than others in general internal medicine; they might not be, because it is very under-diagnosed and very common in all settings. Secondly, I understand you to say, Dr Manu, that there are two discrete populations in your data: patients with idiopathic fatigue and another group where depression is a feature (Table 1 , p 26).
The implication is that patients’ talk about illness is just not valid, whereas physicians’ talk about disease entities is. Patients are giving you their illness experience. What we are doing is to reconstruct that account as a disease. This leaves a certain tension between our different reconstructions: a first-hand yet scientifically suspect patient narrative and a professionally legitimized if experience-distant physician’s tale. White: I would like to continue with the idiopathic group of fatigued subjects, which is in some ways the most interesting.